Healthy bone is constantly being dismantled and rebuilt, which makes bone loss a problem of balance rather than a simple ...

New study reveals how collagen receptors beta1 integrin and DDR2 cooperate to control skeletal progenitor cells for enhanced bone regeneration and healing.

CADD522 preserved fragile bone architecture and cut fat in menopausal mice, but the potential treatment remains years from human use.

Researchers at The University of Texas MD Anderson Cancer Center have uncovered a possible reason why metastatic medullary ...

A new small molecule could treat and prevent excess bone formation. Fibrodysplasia ossificans progressiva (FOP) is an extremely rare disease estimated to affect about one in 2 million people worldwide ...

An experimental compound called AP503 significantly strengthened bones in mice by activating GPR133, a receptor that boosts bone formation while slowing bone loss. Because the same treatment has also ...

A new study of the PhyloBone project of the University of Turku, Finland, identifies hundreds of non-collagenous proteins in the bone matrix that may play regulatory roles in bone formation and ...

Strength training also supports muscle, balance and posture, which lowers the risk of falls.

Scientists have identified a protein that blocks the activity of bone-forming cells (osteoblasts) by stopping them from maturing during the journey to sites of bone formation, a new study has found.

A recent study identifies Neural EGFL-like 2 (NELL2), a secreted protein, as a key regulator of bone homeostasis, offering potential therapeutic applications for osteoporosis. NELL2 promotes ...